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Brain 118:577, 1995 GUARANTORS OF BRAIN: Aids to the Examination of the Peripheral Nervous System, 2nd ed London, Bailliere-Tindall, 1986 ` GUILLAIN-BARRE STUDY GROUP: Plasmapheresis and acute GuillainBarre syndrome Neurology 35:1096, 1985 HADJIVASSILIOU M, CHATTOPADHYAY AK, DAVIES-JONES GA, et al: Neuromuscular disorder as presenting feature of coeliac disease J Neurol Neurosurg Psychiatry 63:770, 1997 HAFER-MACKO CE, SHEIKH KA, LI CY, et al: Immune attack on the Schwann cell surface in acute in ammatory demyelinating polyneuropathy Ann Neurol 39:625, 1996 HAHN AF, BOLTON CF, PILLAY N, et al: Plasma exchange therapy in chronic in ammatory demyelinating polyneuropathy Brain 119: 1055, 1996 HAHN AF, BOLTON CF, ZOCHODNE D, FEASBY TE: Intravenous immunoglobulin treatment in chronic in ammatory demyelinating polyneuropathy Brain 119:1067, 1996 HARDING AE, THOMAS PK: The clinical features of hereditary motor and sensory neuropathy: Types I and II Brain 103:259, 1980 HARDING AE, THOMAS PK: Peroneal muscular atrophy with pyramidal features.

.l(ri-r): l'10351563 J'Qi): 0'gZS 3 . l ( r i * r ): 0 ' 6 3 6 3 2 l8 dif'ference' the to whichcanbe usecl compute fbrward 30.6363281 0.925

J Neurol Neurosurg Psychiatry 47:168, 1984 HART ZH, HOFFMAN W, WINBAUM E: Polyneuropathy, alopecia areata, and chronic lymphocytic thyroiditis Neurology 29:106, 1979 HARTUNG H-P, REINERS K, SCHMIDT B, et al: Serum interleukin-2 concentrations in Guillain-Barre syndrome and chronic idiopathic demyelin ating polyneuropathy: Comparison with other neurological diseases of presumed immunopathogenesis Ann Neurol 30:48, 1991 HARTUNG H-P, STOLL G, TOYKA KV: Immune reactions in the peripheral nervous system, in Dyck PJ, Thomas PK, Grif n JW, et al (eds):.

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In grade IV patients, the outcome is generally dismal, no matter what course is taken, but we have usually counseled against early operation; some neurosurgeons disagree The insertion of ventricular drains into both frontal horns has occasionally raised a patient with severe hydrocephalus to a better grade and facilitated early operation In the.

Aphasia, when it occurs, tends to be a limited form of the Broca type and, in our experience, has been short-lived. With deep small strokes, we have most often encountered incomplete motor syndromes affecting only the arm and hand, without language disturbance or neglect; these are quite dif cult to differentiate from small embolic cortical strokes. The lesions in the corona radiata are larger than typical lacunes (see further on) but probably have a similar pathophysiology. Foix and Levy, who described the clinical effects of deep capsular-basal ganglionic lesions and of more super cial cortical-subcortical ones, found few important differences in the degree and pattern of the hemiplegia and sensory disorder. Homonymous hemianopia may occur with posterior capsular lesions, but it must be distinguished from visual hemineglect of contralateral space. Bilateral cerebral infarctions involving mainly the insular-perisylvian (anterior opercular) regions manifest themselves by a facio-glossopharyngo-masticatory diplegia (anarthria without aphasia; see Mao et al).

19.12 A jet fighter'spositionon an aircraftcarier's runway was timed during landin-u:

6. Let q = S x where q = [ , vn , , h, vd ] . Show that the matrix producing this reordering of the state vector is 1 0 0 0 0 0 0 1 0 0 S = 0 0 0 0 1 and that S 1 = S . 0 1 0 0 0 0 0 0 1 0 7. Use the similarity transformation matrix S to show that the dynamic model for q is 1 0 0 vn 2 0 R+h (R+h) vd vn 0 fd vn vd 2 R+h (R+h) R+h 1 q = 0 0 vn 2 0 q R+h (R+h) 0 0 0 0 1 vn 2 0 2vn fn 0 2GM 3 R+h (R+h)2 (R+h) 0 0 0 cos( ) sin( ) 0 0 0 1 up + (3.103) 2 0 0 0 sin( ) cos( ) 0 where the lines are added to separate the vertical state errors h and vd form the horizontal and tilt errors , vn , and . 8. Consider the unforced (i.e., up = 0), decoupled vertical error dynam2 ics h vd 0 =

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